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By Shamlal Mangray, Ronald A. DeLellis (auth.), Michael A. Blake, Giles Boland (eds.)

Imaging of the adrenal gland has made great growth within the final decade as new applied sciences proceed to conform. Adrenal Imaging highlights the pertinent scientific and pathological details that underpins the exact interpretation and use of adrenal imaging. Written by way of a prestigious workforce of overseas participants, person chapters in Adrenal Imaging function a correct and updated reference of adrenal imaging findings, algorithms and methods in CT, MR nuclear medication, intervention, and trauma. precis sections on the finish of every bankruptcy light up key educating issues to augment retention.

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1992) Detection of novel germline p53 mutations in diverse cancer prone families identified by selecting patients with childhood adrenocortical carcinoma. J Natl Cancer Inst 84:703–710 37. Didolkar MD, Bescher RA, Elias EG, Moore RH (1981) Natural history of adrenal cortical carcinoma: a clinicopathologic study of 42 patients. Cancer 47:2153–2161 38. Weiss LM (1984) Comparative histological study of 43 metastasizing and nonmetastasizing adrenocortical tumors. Am J Surg Pathol 8:163–169 39. Hutter AM, Kayhoe DE (1966) Adrenal cortical carcinoma: clinical features of 138 patients.

Regulation of Hypothalamic-PituitaryAdrenal Axis ACTH, secreted and stored in the basophilic cells of the anterior pituitary gland regulates adrenal cortisol synthesis. The biological half-life of ACTH in the circulation is less than 10 min. ACTH stimulates cyclic AMP, which in turn promotes the synthesis of protein kinase enzymes, thus resulting in the phosphorylation of proteins that activate steroid biosynthesis. ACTH is processed from a large precursor molecule propiomelanocortin (POMC) along with a number of other peptides including b-lipotropin, endorphins and melanocyte stimulating hormone.

Other Etiologies ......................................................... 43 43 .................... 44 Clinical Features of Cushing’s Syndrome S. org Medical Therapies for Cushing’s Syndrome .................................. 50 Other Causes of Increased Mineralocorticoid Action .. Hypoaldosteronism with Low Plasma Renin Activity Apparent Mineralocorticoid Excess .......................... 50 50 50 ...................................................... 50 Hyperaldosteronism with High Plasma Renin Activity Bartter’s Syndrome ....................................................

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